Mouse CCL24 (C-C motif chemokine ligand 24, also known as eotaxin-2 or MPIF-2, macrophage inflammatory protein-4) is a chemokine that regulates recruitment of eosinophils and other immune cells involved in type 2 inflammatory responses in mice (Mus musculus). CCL24 primarily signals through the CCR3 receptor, promoting chemotaxis of eosinophils, basophils, and Th2 lymphocytes to sites of infection, allergic inflammation, or tissue injury. In mice, CCL24 is produced by epithelial cells, macrophages, fibroblasts, and endothelial cells in response to type 2 cytokines such as IL-4 and IL-13, which drive Th2-associated immune responses. Mouse CCL24 plays an important role in experimental models of allergic airway disease (asthma), parasitic helminth infections, and eosinophil-driven inflammation, where it contributes to recruitment of eosinophils into lung and mucosal tissues. While CCL24-mediated eosinophil trafficking supports protective immunity against parasites and participates in tissue repair, excessive or prolonged expression can promote chronic allergic inflammation and tissue remodeling. Because mice are widely used as genetic models for studying allergic disease, parasite immunity, and Th2-mediated inflammation, characterization of mouse CCL24 has been important for understanding CCR3-dependent eosinophil recruitment and developing therapeutic strategies targeting eosinophilic inflammatory disorders.